Molecular Biotechnology Engeenier, Faculty of Science, Universidad de Chile (2004)
Ph.D Pharmacology. Universidad de Chile, Santiago, Chile (2011)
LINEAS DE INVESTIGACIÓN
Aging and wound healing
PUBLICACIONES:
Saldías MP, Morgan A, Fernández C, Díaz C, Silva A, Jaña F, Gómez, Briceño F, Oyarzún A, Maldonado F, Cerda O, Smith PC, Cáceres C. Aged blood factors decrease cellular responses associated with delayed gingival wound repair. PloS One,2017. 12(9), e0184189.
Retamal IN, Hernandez R, González-Rivas C, Cáceres M, Arancibia R, Romero A, Matinez J, Smith PC. (2016).Methylglyoxal and methylglyoxal-modified collagen as inducers of cellular injury in gingival connective tissue cells. J Periodontal Res 51(6):812-821.
Caceres, M., Ortiz, L., Recabarren, T., Romero, A., Colombo, A., Leiva-Salcedo, E., Varela, D., Rivas, J., Silva, I., Morales, D., Campusano, C., Almarza, O., Simon, F., Toledo, H., Park, K.S., Trimmer, J.S., Cerda, O. TRPM4 Is a Novel Component of the Adhesome Required for Focal Adhesion Disassembly, Migration and Contractility. PLoS One 10 (2015), e0130540. First author
Smith, P. C., Martínez, C., Cáceres, M., & Martínez, J. (2015). Research on growth factors in periodontology. Periodontology 2000, 67(1), 234–250. Impact factor 4.04. Ranking 3/90 (dentistry).
Smith, P. C., Cáceres, M., Martínez, C., Oyarzún, A., & Martínez, J. (2015). Gingival Wound Healing: An Essential Response Disturbed by Aging? Journal of Dental Research. 94(3), 395–402. Impact factor 4.6. Ranking 2/90 (dentistry)
Cerda, O., Cáceres, M., Park, K.-S., Leiva-Salcedo, E., Romero, A., Varela, D., et al. (2015). Casein kinase-mediated phosphorylation of serine 839 is necessary for basolateral localization of the Ca2+- activated non-selective cation channel TRPM4. Pflügers Archiv – European Journal of Physiology. doi:10.1007/s00424-014-1610-3
Cáceres, M., Oyarzún, A., & Smith, P. C. (2014). Defective Woundhealing in Aging Gingival Tissue. J Dent Res 93(7):691-697. Corresponding autor Impact factor 4.6 Ranking 2/90 (dentistry)Sarmiento D, Montorfano I, Cáceres M, Echeverría C, Fernández R, Cabello-Verrugio C, Cerda O, Tapia P, Simon F. Endotoxin-induced vascular endothelial cell migration is dependent on TLR4/NF-κB pathway, NAD(P)H oxidase activation, and transient receptor potential melastatin 7 calcium channel activity. Int J Biochem Cell Biol. 2014 Oct;55:11-23.